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glutathione epilepsy Neuron–glia interactions in the pathophysiology of JCI Insight - REST
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glutathione epilepsy Neuron–glia interactions in the pathophysiology of JCI Insight - REST
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glutathione epilepsy Neuron–glia interactions in the pathophysiology of JCI Insight - REST
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glutathione epilepsy Neuron–glia interactions in the pathophysiology of JCI Insight - REST
JCI Insight - REST contributes to AKI-to-CKD transition through inducing ferroptosis in renal tubular epithelial cells
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GSH depletion cause indirectly formation of endotoxins in the cytoplasm of pigmented SN cells that contribute to the degeneration of these neurons in PD “Figure 2.” As previously mentioned the activity of γ-GT is up-regulated significantly in the parkinsonian SN [61]
glutathione epilepsy Neuron–glia interactions in the pathophysiology of JCI Insight - REST
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