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FIGURE 7 In summary, PCs participate in T2DM pathophysiology via multiple mechanisms, such as enhancing insulin secretion, safeguarding pancreatic β-cells, optimizing mitochondrial function and insulin resistance, and regulating hepatic lipid metabolism

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Curcumin, demathoxycurcumin, bis-demethoxycumin, and calebin-A, all extracted from Curcuma longa, have been shown to protect PC12 cells from β-amyloid assault (Maurer et al., 1997