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Vice versa, confronted with the permanent central venous occlusion, the therapy effect is starting centrally, from the occluded superior sagittal sinus, as a prime noxious event (note, instead increased positive pressure, BPC 157-rats presented the negative pressure values close to those noted in the superior sagittal sinus of the healthy rats)

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The dramatic efficacy gap between rodent and human studies likely reflects bioavailability differences rather than species-specific receptor variations