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This alters the molecular structure, cleaving the peptide bonds and rendering the compound completely useless for precise in-vitro laboratory applications
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VEGF-A signals through VEGFR2 (KDR/Flk-1) on endothelial cells, activating PLCγ-PKC-MEK-ERK and PI3K-Akt-eNOS pathways that collectively drive endothelial proliferation, migration, and tube formation
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