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Since no clear loss of 2a activity was observed in PSMA-negative cells, we further blocked PSMA-mediated uptake of 2a in LNCaP cells: either by saturating the receptor with excess PSMA-Cbz , or by siRNA-mediated knockdown of folh1
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Effects of once-weekly dosing of a long-acting release formulation of exenatide on glucose control and body weight in subjects with type 2 diabetes
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